Age-related Macular Degeneration
Pathology
Advanced AMD is classified as
Neovascular AMD
Atrophic AMD
Neovascular AMD is characterised by neovascular proliferation underneath the neuroretina. This is also called choroidal neovascularisation.
Atrophic AMD is characterised by atrophy of the RPE and overlying neurosensory retina. This is also called geographic atrophy
Choroidal neovascularisation and geographic atrophy may develop in the same eye, because they are on the same continuum of disease activity
In addition to choroidal neovascularisation, there are two other forms of AMD-associated neovascularisation - polypoidal choroidal vasculopathy - retinal angiomatous proliferation
Neovascular amd is responsible for the majority of severe vision loss
The fluid in macular degeneration can be due to leaky blood vessels or haemorrhage
Reference: Ape RS 2021 in NEJM
Diagnosis
Epidemiology (the patient)
Non-modifiable risk factors
Age
Genetics (strong predisposition)
Appears to affect men and women similarly
Modifiable risk factors
Uncontrolled hypertension
Smoking
Being overweight
The presentation
Painless vision loss
Progressive, particularly of central vision
Can be acute in the context of neovascular AMD (e.g. blood or fluid accumulation)
Establishing the diagnosis
Historically, angiography with fluorescein or indocyanine was used to assess the choroidal or retinal vasculature
Indocyanine was particularly useful for identifying the choroidal vasculature for polypoidal choroidal vasculopathy or retinal angiomatous proliferation
This has now largely been replaced by optical coherence tomography
Fundus autofluorescence is useful to assess the health of the retinal pigment epithelium
Identifying polypoidal choroidal vasculopathy is important because when present, treatment with photodynamic therapy may be beneficial
Reference: Ape RS 2021 in NEJM
Management
Address modifiable risk factors:
Uncontrolled hypertension
Smoking
Being overweight
For intermediate severity, that is, before geographic atrophy of choroidal neovascularisation, there is some evidence that oral supplementation may slow disease progression.
Age related eye disease studies (AREDS)
AERDS I supplementation protocol included:
Vitamin C 500 mg
Zinc 80 mg with Copper 2 mg (added to avoid zinc-related copper deficiency)
Vitamin E 400 IU
Beta carotene 15 mg
Beta carotene has been associated with an increased risk of lung cancer amongst current or past smokers
In the age related eye disease study 2 (AREDS II), beta carotene was replaced with
Lutein 10 mg
Zeaxanthin 2 mg
So the the AREDS II formula is:
Vitamin C 500 mg
Zinc 80 mg with Copper 2 mg (added to avoid zinc-related copper deficiency)
Vitamin E 400 IU
Lutein 10 mg
Zeaxanthin 2 mg
Reference: Ape RS 2021 in NEJM
Polypoidal choroidal vasculopathy
Photodynamic therapy is a treatment modality for polypoidal choroidal vasculopathy
Photodynamic therapy involves an intravenous injection of verteporfin, a compound which can be photoactivated and selectively binds to abnormal blood vessels. Verteporfin is then activated with a lower power laser to induce vascular regression
a combination of photodynamic therapy and anti-VEG thearpy results in greater gain in visual acuity in polypoidal choroidal vasculopathy. This benefit has been observed when photodynamic therapy is used in conjunction to ranibizumab but not with aflibercept
Reference: Ape RS 2021 in NEJM
Patient Information
What do I have?
Age related macular degeneration
The macula is the central part of your vision
With age, you get a build up of debris in the layer just under the special sensory cells of your eye
This debris is then associated with death of the supporting and special sensory cells in the area, causing loss of vision
You can also have abnormal blood vessels grow, which then cause bleeding and swelling that clouds your vision
Ref: Fleckenstein 2024 JAMA review
Why me?
It is unclear exactly what causes age related macular degeneration
Research shows that some things are associated with an increased risk of developing macular degeneration. Some of these are modifiable, and others are not
Non-modifiable risk factors include
Getting older (age)
Genetic predisposition (strong)
Modifiable risk factors include
Having high (uncontrolled) blood pressure
Smoking
Being overweight
Reference: Ape RS 2021 in NEJM